74 Studien
Forschungsbibliothek
Peer-reviewed Papers aus Top-Journals, zusammengefasst und nach Evidenzstärke bewertet. Updates jeden Mo, Mi & Fr.
15.–21. Mär 2026
2Two Opposing Brain Fuel Patterns May Predict Who Keeps Their Cognition With Age
Brain white matter (the wiring that connects brain regions) uses glucose differently depending on where you look. In over 3,000 participants across two studies, higher glucose use in expected areas like the corpus callosum linked to better thinking skills. But higher glucose use in unusual areas like the corona radiata linked to worse cognition, likely a sign the brain is compensating. Over time, people with strong "normal" metabolism and low "compensatory" metabolism declined more slowly.
How Excess Fructose May Damage Far More Than Just Your Liver
This review pulls together evidence showing fructose does more than add calories. It triggers a chain reaction: uric acid buildup, mitochondrial stress, and fat storage signals that affect the liver, kidneys, pancreas, gut, heart, lungs, and brain. The damage traces back to how fructose is processed differently than glucose, depleting cellular energy and driving inflammation. Animal and human studies both point to fructose overload as a metabolic disruptor across nearly every organ system.
8.–14. Mär 2026
3Blood Proteins May Reveal Two Critical Windows for Frailty Around Ages 50 and 63
A study of over 50,000 UK Biobank participants found 1,339 blood proteins linked to frailty. Researchers built a "proteomic frailty score" that predicted risk for 199 diseases and responded to 84 modifiable risk factors. The most striking finding: frailty-related protein changes showed two distinct peaks, around ages 50 and 63. These windows could represent key moments when biological aging accelerates.
Caloric Restriction Slows Aging Most in the Heart and Metabolism
Cutting calories doesn't slow aging evenly across all organs. In a two-year trial, 185 adults were randomly assigned to caloric restriction or normal eating. The caloric restriction group aged about one year less in their cardiovascular and metabolic systems over 24 months. Kidney aging, however, didn't budge, and liver aging only slowed modestly at the two-year mark.
Vitamin C May Slow Primate Aging by Blocking Iron-Driven Cell Damage
As primates age, iron builds up in tissues and fuels a chain reaction of fat damage in cells. Researchers call this process "ferro-aging" and found that a specific enzyme (ACSL4) drives it. When aged monkeys received vitamin C for over 40 months, it directly blocked that enzyme. The result was reduced tissue damage, better brain and metabolic function, and biological age clocks that ticked backward across multiple organs.
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